Rivaroxaban
- A new oral direct factor Xa inhibitor approved for stroke prevention in atrial fibrillation (20mg daily/15mg daily in patients with Clcr 15-50) and for prevention of DVT/PE in patients undergoing total hip or knee replacement (10mg daily)
Relevant Clinical Trials
Atrial Fibrillation
Hip Replacement
- Eriksson BI, et al. Rivaroxaban versus enoxaparin for thromboprophylaxis after hip arthroplasty. New Engl J Med 2008; 358:2765-75.
- Kakkar AK, et al (2008). Extended duration rivaroxaban versus short term enoxaparin for the prevention of venous thromboembolism after total hip arthroplasty: a double-blind, randomised controlled trial. Lancet 2008; 372:31-9.
Knee Replacement
- Lassen MR, et al. Rivaroxaban vs. enoxaparin for thromboprophylaxis after total knee arthroplasty. New Engl J Med 2008; 358:2776-86.
- Turpie AG et al. Rivaroxaban versus enoxaparin for thromboprophylaxis after total knee replacement arthroplasty (RECORD 4): a randomized trial. Lancet 2009; 373: 1673-80.
Medication Guide
Patient Education Documentation
- Coming soon
Clcr |
Recommended |
Recommended Dose in Ortho Surgery |
AUC |
T 1/2 |
> 80 ml/min |
20mg qday |
10mg daily |
- |
8.3 hrs |
50 – 80 ml/min |
1.4 x increase |
8.7 hrs |
||
30 – 50 ml/min |
Clcr 15-50 ml/min: Clcr< 15 ml/min: |
1.5 x increase |
9.0 hrs |
|
< 30 ml/min |
Clcr < 30: |
1.6 x increase |
9.5 hrs |
warfarin to rivaroxaban |
stop warfarin and start rivaroxaban when INR < 2 |
rivaroxaban to warfarin |
start warfarin and stop rivaroxaban 3 days later |
LMWH/ fondaparinux to rivaroxaban |
stop parenteral anticoagulant and administer rivaroxaban 0-2 hours before next parenteral dose would have been given |
IV heparin to rivaroxaban |
administer first dose of rivaroxaban at time of discontinuation |
rivaroxaban to parenteral anticoagulant |
Stop rivaroxaban and administer first dose of parenteral anticoagulant at the time the next dose of rivaroxaban would have been given |
Time of last dose of rivaroxaban |
|
Standard Risk of Bleeding |
High Risk of Bleeding |
24 hrs |
24-48 hours |
Pharmacodynamic Interactions
The concurrent use of rivaroxaban with other anticoagulants, antiplatelet agents, and nonsteroidal anti-inflammatory agents is expected to increase the risk of bleeding in comparison to use of rivaroxaban alone.
Pharmacokinetic Interactions
The absorption of rivaroxaban is mediated by P-glycoprotein (P-gp). P-gp inhibitors can increase the absorption of rivaroxaban, increasing both AUC and Cmax. Conversely, P-gp inducers can reduce the absorption of rivaroxaban, decreasing AUC and Cmax.
The metabolism of rivaroxaban is mediated by CYP3A4. CYP3A4 inhibitors can decrease the metabolism of rivaroxaban, increasing both AUC and Cmax. Conversely, CYP3A4 inducers can increase the metablism of rivaroxaban, decreasing AUC and Cmax
Agents that interfere with both P-gp and CYP3A4 are likely to cause more significant interactions with rivaroxaban than agents that interfere with P-gp or CYP3A4 alone.
Drug Class |
Examples |
Known or Probable Effect |
US PI Recommendation |
Suggested Management Guidelines |
Combined P-gp inhibitor and CYP3A4 inhibitor |
amiodarone, atazanavir azithromycin, boceprevir, clarithromycin, conivaptan, cyclosporine, darunavir, delavirdine, diltiazem, dronedarone, erythromycin, fluconazole, fosamprenavir,imatinib,indinavir, |
Significant increase in rivaroxaban concentration |
1. Avoid use of P-gp/strong CYP3A4 inhibitors (eg: ketoconazole, intraconazole, ritonavir, conivaptan) . 2. Use P-gp/moderate CYP3A4 inhibitors (eg: amiodarone, azithromycin, diltiazem, dronedarone, erythromycin, felodipine, , quinidine, ranolazine, verapamil) with caution in patients with renal impairment 3. No precautions necessary for use of clarithromycin and erythromycin in patients receiving rivaroxaban specifically for DVT prophylaxis |
Avoid use Risk higher in pts with renal impairment |
Combined P-gp inducer and 3A4 inducer |
barbiturates, carbamazepine, phenytoin, rifampin, St Johns wort |
Significant reduction in rivaroxaban concentration |
Avoid use of P-gp/strong CYP3A4 inducers (eg: carbamazepine, phenytoin, rifampin, St Johns wort) |
Avoid use |
Other inducers of P-gp |
doxorubicin, prazosin, tipranavir, trazodone, vinblastine |
Significant reduction in rivaroxaban concentration |
Not specifically addressed |
Avoid use |
Other inducers of CYP3A4 |
bosentan, efavirenz, etravirine, fosphenytoin, nafcillin, nevirapine, oxarbazepine, primidone, rifabutin, rifapentine |
Significant reduction in rivaroxaban concentration |
Not specifically addressed |
Avoid use |
Rivaroxaban is not intended to be monitored using routine coagulation testing. Its fixed dosing is not intended to be adjusted on the basis of any coagulation laboratory parameter. In certain clinical situations in which the absence of anticoagulant effect induced by rivaroxaban needs to be assured, prothrombin time (PT) and partial thromboplastin time (PTT) can be evaluated and should be normal.
These settings include:
- to assure appropriate rivaroxaban clearance prior to invasive procedures
- to assure appropriate rivaroxaban clearance prior to thrombolytic therapy for acute ischemic stroke
NOTE: there is no reliable correlation between elevated PT/PTT and therapeutic effect
There is NO REVERSAL AGENT OR ANTIDOTE for rivaroxaban. Very limited data, primarily non-human, are available to guide management of bleeding.
Mild Bleeding
- delay next dose or discontinue therapy
Moderate to Severe Bleeding
- symptomatic treatment
- mechanical compression
- surgical intervention
- fluid replacement and hemodynamic support
- blood product transfusion
- oral charcoal (if rivaroxaban administered < 2 hrs prior)
- NOTE: not dialyzable
Life Threatening Bleeding
- measures above
- charcoal filtration
- last resort: rVIIa or PCC